Salicylic acid and jasmonic acid signal through a noncanonical pathway to promote effector-triggered immunity
X. DONG (1), L. Liu (1), F. Sonbol (2), Y. Gu (3), J. Withers (3), J. Yao (4) (1) Duke University, U.S.A.; (2) Sinai University, Egypt; (3) Duke University, U.S.A.; (4) Western Michigan University, U.S.A.

It is an evolutionary conundrum how plants evolved effector-triggered immunity (ETI) as a major defense mechanism against biotrophic pathogens, as ETI-associated programmed cell death may leave them vulnerable to necrotrophic pathogens that thrive on dead host cells. Interestingly, during ETI, the normally antagonistic defence hormones, salicylic acid (SA) against biotrophs and jasmonic acid (JA) against necrotrophs, both accumulate to high levels. In my presentation, I will report the unique interplay between SA and JA discovered in my lab, which solves this apparent cell-death paradox of ETI.

Abstract Number: P5-2
Session Type: Plenary